The smart Trick of apl11 That Nobody is Discussing
The smart Trick of apl11 That Nobody is Discussing
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promyelocytic (M3) acute leukaemia; quite a few clients express an abnormal morphologic spectrum intermediate concerning M2 and M3 AML.
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large level of regular or dystrophic promyelocytes in peripheralblood and in bone marrow; no intracytoplasmic Auer rods; myeloperoxydasereaction positive; immunocytochemical detection having an anti-PLZF exhibits adistinct punctate nuclear distribution of the protein, suggesting itscompartmentalization from the nucleus.
Speedy detection of prognostically sizeable fusion transcripts in acute leukemia employing simplified multiplex reverse transcription polymerase chain response.
ZBTB16-RARα variant of acute promyelocytic leukemia with tuberculosis: a scenario report and evaluation of literature.
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Profitable allogeneic bone marrow transplantation in a very circumstance of variant acute promyelocytic leukemia with ZBTB16-RARA.
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A situation of acute promyelocytic leukaemia with unusual cytological attributes along with a ZBTB16-RARA fusion gene.
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Characterization of cryptic rearrangements and variant translocations in acute promyelocytic leukemia.
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the translocation requires a breakpoint from the zinc fingerregion of PLZF, with fusion of two zinc fingers towards the RARa B region toform a 5 PLZF - 3 RARa fusion gene; the reciprocal 5 RARa - three PLZF genefuses 7 zinc fingers to the RARa region; RARas breakpoint occurs in1- because of thealternative splicing of PLZF gene, two kinds of PLZF-RARa protein is usually detected: